The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 40
VITAMIN K IN FOOD 1 . NORMAL PROTHROMBIN FORMATION AMINO -
ACIDURIA MILLON ' S REACTION FOR TYROSINE PROTEIN IN FOOD PAPER
CHROMATOGRAM VA - PROTHROMBIN 2 PROTHROMBIN FORMED IN
LIVERCLOT ...
VITAMIN K IN FOOD 1 . NORMAL PROTHROMBIN FORMATION AMINO -
ACIDURIA MILLON ' S REACTION FOR TYROSINE PROTEIN IN FOOD PAPER
CHROMATOGRAM VA - PROTHROMBIN 2 PROTHROMBIN FORMED IN
LIVERCLOT ...
Page 66
However , the formation of connective tissue membranes around the portal tracts
, as a result of inflammatory reactions partially secondary to liver cell injury , are
more important . These membranes extend from the tracts into the parenchyma ...
However , the formation of connective tissue membranes around the portal tracts
, as a result of inflammatory reactions partially secondary to liver cell injury , are
more important . These membranes extend from the tracts into the parenchyma ...
Page 172
Protein formation takes place on ribosomes that are free in the cytoplasm and
also on those attached to the endoplasmic reticulum . They consist of
ribonucleoprotein ( RNP ) particles with a molecular weight of approximately 2 ,
000 , 000 .
Protein formation takes place on ribosomes that are free in the cytoplasm and
also on those attached to the endoplasmic reticulum . They consist of
ribonucleoprotein ( RNP ) particles with a molecular weight of approximately 2 ,
000 , 000 .
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall