The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 15
Before or immediately after passing behind the duodenum , it gives origin to the
posterior superior pancreaticoduodenal artery ( the retroduodenal artery of
Michels ) . Its origin is often hidden by dense fibrous tissue , and , passing to the
right ...
Before or immediately after passing behind the duodenum , it gives origin to the
posterior superior pancreaticoduodenal artery ( the retroduodenal artery of
Michels ) . Its origin is often hidden by dense fibrous tissue , and , passing to the
right ...
Page 47
This pigment , soluble in lipids but only slightly soluble in water , gives the red
diazo reaction ( with sodium nitrite and sulfanilic acid ) of van den Bergh ;
however , this is possible only after special treatment of the pigment to increase
its water ...
This pigment , soluble in lipids but only slightly soluble in water , gives the red
diazo reaction ( with sodium nitrite and sulfanilic acid ) of van den Bergh ;
however , this is possible only after special treatment of the pigment to increase
its water ...
Page 83
... with little milk or cheese consumption , whereas it is a frequent finding in
animals , e . g . , horses , which have been immunized over long periods of time
against diphtheria or tetanus . Amyloid deposit gives the liver a dry , waxy
appearance .
... with little milk or cheese consumption , whereas it is a frequent finding in
animals , e . g . , horses , which have been immunized over long periods of time
against diphtheria or tetanus . Amyloid deposit gives the liver a dry , waxy
appearance .
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall