A Compilation of paintings on the normal and pathologic anatomy of the digestive system: Liver, biliary tract and pancreas, Volume 3, Part 3 |
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Page 48
Increased production or decreased excretion of biliary pigments leads to
jaundice without bile flow impairment. Hemolytic jaundice is due to
overproduction of bile pigment. It occurs in septicemia, pernicious anemia,
acquired or congenital ...
Increased production or decreased excretion of biliary pigments leads to
jaundice without bile flow impairment. Hemolytic jaundice is due to
overproduction of bile pigment. It occurs in septicemia, pernicious anemia,
acquired or congenital ...
Page 74
Ascites is well correlated with diminished albumin synthesis in all types of
cirrhosis, but, setting up a vicious circle, the albumin loss into the ascitic fluid also
increases the hypo-albuminemia. Sodium retention, as it may be brought on by
heart ...
Ascites is well correlated with diminished albumin synthesis in all types of
cirrhosis, but, setting up a vicious circle, the albumin loss into the ascitic fluid also
increases the hypo-albuminemia. Sodium retention, as it may be brought on by
heart ...
Page 87
This is associated with increased copper deposition in various organs,
particularly in the liver, brain, kidney and Descemct's membrane of the cornea.
The excess deposition in tissue is considered injurious and probably is
responsible for the ...
This is associated with increased copper deposition in various organs,
particularly in the liver, brain, kidney and Descemct's membrane of the cornea.
The excess deposition in tissue is considered injurious and probably is
responsible for the ...
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Contents
Part | 1 |
PLATE PAGE NUMBER NUMBER 1 Development of Liver and Its Venous System | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
133 other sections not shown
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Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood capillaries carcinoma cause celiac cent cholecystitis cholestasis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enlarged enzymes esophageal esophageal varices excretion extrahepatic fibers fibrosis fistula flocculation formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular histologic increased infection inferior injury intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobule lymphatic metabolism metastases necrosis nodes normal organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules renal result right hepatic septa serum shunt sinusoids sphincter spleen splenic stones surface surgical tion tumor urobilinogen usually varices vascular vena cava vessels viral hepatitis wall
Bibliographic information
| Title | A Compilation of paintings on the normal and pathologic anatomy of the digestive system: Liver, biliary tract and pancreas, Volume 3, Part 3 A Compilation of Paintings on the Normal and Pathologic Anatomy of the Digestive System: Liver, Biliary Tract and Pancreas, Frank Henry Netter |
| Authors | Frank Henry Netter, Ciba Pharmaceutical Company |
| Editor | Ernst Oppenheimer |
| Edition | 2 |
| Publisher | Ciba Pharmaceutical, 1964 |
| Original from | the University of Michigan |
| Digitized | 1 Oct 2010 |
| ISBN | 0914168053, 9780914168058 |
| Length | 200 pages |
| Subjects | › Biliary tract Digestive organs Liver Medical / Gastroenterology Pancreas |
| Export Citation | BiBTeX EndNote RefMan |