The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 43
However , other evidence indicates that under abnormal circumstances ,
especially intra - or extrahepatic cholestasis , the liver forms excessive alkaline
phosphatase and releases it to the blood . Whatever the physiologic basis , in
almost all ...
However , other evidence indicates that under abnormal circumstances ,
especially intra - or extrahepatic cholestasis , the liver forms excessive alkaline
phosphatase and releases it to the blood . Whatever the physiologic basis , in
almost all ...
Page 51
... FOLLOWING TESTS YIELD RESULTS AS INDICATED : ASSUMED WHEN
ONE OR MORE OF FOLLOWING TESTS ... terms has recently been changed on
the basis of electron microscopic evidence , which indicates that interference with
...
... FOLLOWING TESTS YIELD RESULTS AS INDICATED : ASSUMED WHEN
ONE OR MORE OF FOLLOWING TESTS ... terms has recently been changed on
the basis of electron microscopic evidence , which indicates that interference with
...
Page 53
If by that time the dye does not appear , it indicates hepatocellular damage . ...
crystals ( square or rhomboid plates with clipped edges ) and the lustrous
clusters of granular calcium bilirubinate indicate tendency for gallstone formation
.
If by that time the dye does not appear , it indicates hepatocellular damage . ...
crystals ( square or rhomboid plates with clipped edges ) and the lustrous
clusters of granular calcium bilirubinate indicate tendency for gallstone formation
.
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall