The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 41
Provided intestinal absorption is not increased as in hyperthyroidism or
decreased as in deficiency diseases , and provided renal insufficiency does not
interfere , a large amount of galactose in the urine may be considered an
indication of liver ...
Provided intestinal absorption is not increased as in hyperthyroidism or
decreased as in deficiency diseases , and provided renal insufficiency does not
interfere , a large amount of galactose in the urine may be considered an
indication of liver ...
Page 88
The hydrochloric acid of the stomach reduces the ferric to the ferrous form , which
passes through the intestinal mucosa . There it is reoxidized , and the ferric iron is
bound to a protein , apoferritin . The iron - protein complex , ferritin , is the ...
The hydrochloric acid of the stomach reduces the ferric to the ferrous form , which
passes through the intestinal mucosa . There it is reoxidized , and the ferric iron is
bound to a protein , apoferritin . The iron - protein complex , ferritin , is the ...
Page 102
INTESTINAL FOCUS 3 . ... without an intermediate host , by water , food , etc . ,
which has been in contact with fecal human material , enters the intestinal tract by
the mouth , passes the stomach and loses its cystic wall in the small intestine .
INTESTINAL FOCUS 3 . ... without an intermediate host , by water , food , etc . ,
which has been in contact with fecal human material , enters the intestinal tract by
the mouth , passes the stomach and loses its cystic wall in the small intestine .
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall