The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 58
Disturbances of this development lead to an irregular arrangement of the ducts ,
resulting in solid nodules or in cysts . ... The same embryologic disturbance leads
to cyst formation when the hamartomatous cavities become large or ...
Disturbances of this development lead to an irregular arrangement of the ducts ,
resulting in solid nodules or in cysts . ... The same embryologic disturbance leads
to cyst formation when the hamartomatous cavities become large or ...
Page 108
THROMBOSIS OF PORTAL VEIN VASCULAR DISTURBANCES II Portal Vein G
CIBA Sudden complete obstruction of the portal vein and its branches by a
thrombus leads to a clinically dramatic picture dominated by hematemesis ,
melena ...
THROMBOSIS OF PORTAL VEIN VASCULAR DISTURBANCES II Portal Vein G
CIBA Sudden complete obstruction of the portal vein and its branches by a
thrombus leads to a clinically dramatic picture dominated by hematemesis ,
melena ...
Page 126
In the presence of a large stone , the color and consistency of the bile are usually
altered . The accompanying inflammation and cystic duct obstruction lead to
admixture of mucus and reduction of the bile pigment content . Cholelithiasis may
or ...
In the presence of a large stone , the color and consistency of the bile are usually
altered . The accompanying inflammation and cystic duct obstruction lead to
admixture of mucus and reduction of the bile pigment content . Cholelithiasis may
or ...
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall