The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 38
Abnormalities of serum proteins almost always reflect a far more general
disturbance of liver functions in spite of the fact that the liver has a monopoly to
manufacture albumin , fibrinogen , prothrombin and the greater part of the
globulins .
Abnormalities of serum proteins almost always reflect a far more general
disturbance of liver functions in spite of the fact that the liver has a monopoly to
manufacture albumin , fibrinogen , prothrombin and the greater part of the
globulins .
Page 56
SERUM AMYLASE SERUM LIPASE COMPOSITION OF STOOL LIBERATION OF
GLUCOSE FROM STARCH HYDROLYSIS OF OLIVE OIL FAT NORMAL % OF
INTAKE EXCRETED IN FECES 100 200 gm . INTAKE / DAY NORMAL SLIGHT ...
SERUM AMYLASE SERUM LIPASE COMPOSITION OF STOOL LIBERATION OF
GLUCOSE FROM STARCH HYDROLYSIS OF OLIVE OIL FAT NORMAL % OF
INTAKE EXCRETED IN FECES 100 200 gm . INTAKE / DAY NORMAL SLIGHT ...
Page 186
... 179 TPNH , I72 , 173 droplets increased in , 171 scintigram , 176
transaminases ( see also serum vacuoles in , 174 , 177 self - perpetuation , 182
transaminase ) , 172 , 173 poisons , 171 , 181 serum albumin ( see also albumin
) , transferase ...
... 179 TPNH , I72 , 173 droplets increased in , 171 scintigram , 176
transaminases ( see also serum vacuoles in , 174 , 177 self - perpetuation , 182
transaminase ) , 172 , 173 poisons , 171 , 181 serum albumin ( see also albumin
) , transferase ...
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall