A Compilation of paintings on the normal and pathologic anatomy of the digestive system: Liver, biliary tract and pancreas, Volume 3, Part 3 |
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Page 82
Intrinsic obstructive tumors are usually malignant, represented by either cancer of
the biliary ducts or cancer of the papilla of Vater. Carcinoma of the pancreas (see
page 148), which may compress or kink the ducts, extension of carcinoma of ...
Intrinsic obstructive tumors are usually malignant, represented by either cancer of
the biliary ducts or cancer of the papilla of Vater. Carcinoma of the pancreas (see
page 148), which may compress or kink the ducts, extension of carcinoma of ...
Page 93
The incubation period of homologous serum hepatitis may be very long, usually
more than 40 days. The virus appears in the feces. Gamma globulin provides no
protection. The serum hepatitis has no preference for age periods. The clinical ...
The incubation period of homologous serum hepatitis may be very long, usually
more than 40 days. The virus appears in the feces. Gamma globulin provides no
protection. The serum hepatitis has no preference for age periods. The clinical ...
Page 132
Bile Duct Fistulae A cholecystoduodenal fistula is usually the result of acute
cholecystitis with obstruction of the gallbladder neck by a solitary stone too large
to pass the cystic duct (see page 127) and a subsequent pericholecystitis (see
page ...
Bile Duct Fistulae A cholecystoduodenal fistula is usually the result of acute
cholecystitis with obstruction of the gallbladder neck by a solitary stone too large
to pass the cystic duct (see page 127) and a subsequent pericholecystitis (see
page ...
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Contents
Part | 1 |
PLATE PAGE NUMBER NUMBER 1 Development of Liver and Its Venous System | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood capillaries carcinoma cause celiac cent cholecystitis cholestasis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enlarged enzymes esophageal esophageal varices excretion extrahepatic fibers fibrosis fistula flocculation formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular histologic increased infection inferior injury intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobule lymphatic metabolism metastases necrosis nodes normal organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules renal result right hepatic septa serum shunt sinusoids sphincter spleen splenic stones surface surgical tion tumor urobilinogen usually varices vascular vena cava vessels viral hepatitis wall