The Ciba Collection of Medical Illustrations: HeartThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
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Page 58
... waves in lead I are small , but they are tall in leads II , III , and aVF , often exceeding the upper limit of nor- mal ( 2.5 mm ) for lead II . The vector loop is down , forward , and large . Mod- erately tall P waves are present in ...
... waves in lead I are small , but they are tall in leads II , III , and aVF , often exceeding the upper limit of nor- mal ( 2.5 mm ) for lead II . The vector loop is down , forward , and large . Mod- erately tall P waves are present in ...
Page 59
... waves , relative to the normal , in leads V , and V2 , and deep S waves in leads V , and V. The R / S amplitude ratio in lead V1 is abnormal , indicating a tall R wave with respect to the depth of the S wave . Normally , this ratio ...
... waves , relative to the normal , in leads V , and V2 , and deep S waves in leads V , and V. The R / S amplitude ratio in lead V1 is abnormal , indicating a tall R wave with respect to the depth of the S wave . Normally , this ratio ...
Page 66
... wave ) which appears different from the P waves of the basic mechanism and is premature in time . This is the atrial premature contraction . Measure the time interval from the premature P wave to the P wave immediately in front , and ...
... wave ) which appears different from the P waves of the basic mechanism and is premature in time . This is the atrial premature contraction . Measure the time interval from the premature P wave to the P wave immediately in front , and ...
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Common terms and phrases
action activity acute anterior aorta aortic aortic valve appearance arch associated atrial atrioventricular become block blood body border branch bundle cardiac cardiomegaly cause cells changes CIBA circulation clinical common complete Continued contraction coronary artery CUSP decrease defect diastolic dilatation direction disease drugs early effects endocardial enlargement failure fibers flow heart hypertension important increase infarction INFERIOR insufficiency INTERNAL interventricular involved lateral lead left atrium left ventricle left ventricular lesions lung mechanism membrane mitral valve murmur muscle myocardial myocardial infarction NERVE Netter node normal occur opening organ origin output oxygen patients peripheral PLATE portion position posterior present pressure projection PULMONARY ARTERY result rheumatic fever right atrium right ventricle right ventricular seen segment septal septum severe shows side sinus sound stenosis SUPERIOR sympathetic systolic thoracic tion tissue tricuspid TRUNK upper usually VEIN VENA CAVA venous vessels wall waves