The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 38
... abnormal . After adding to small amounts of serum a solution containing ammonium sulfate and sodium chloride , a turbidity devel- ops , the degree of which is read , either optically or spectrophotometrically , after an interval of 30 ...
... abnormal . After adding to small amounts of serum a solution containing ammonium sulfate and sodium chloride , a turbidity devel- ops , the degree of which is read , either optically or spectrophotometrically , after an interval of 30 ...
Page 51
... abnormal results of the cephalin flocculation and increase of urinary uro- bilinogen and Bromsulphalein reten- tion may disclose evidence of damage otherwise not available . However , a nor- mal outcome of these tests does not exclude ...
... abnormal results of the cephalin flocculation and increase of urinary uro- bilinogen and Bromsulphalein reten- tion may disclose evidence of damage otherwise not available . However , a nor- mal outcome of these tests does not exclude ...
Page 58
... abnormal , and values lower than 25 or greater than 185 units are probably abnormal . Values below 50 units are encountered in 30 to 40 per cent of patients with diseases of the liver and bile ducts , acute cholecystitis , pneumonia ...
... abnormal , and values lower than 25 or greater than 185 units are probably abnormal . Values below 50 units are encountered in 30 to 40 per cent of patients with diseases of the liver and bile ducts , acute cholecystitis , pneumonia ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall