The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 92
... abscess formation ( purulent hepatitis ) , which may involve the portal vein branches to produce pylephlebitic abscesses , with further throm- botic extension along the portal vein tree . But the pylephlebitic abscesses may develop from ...
... abscess formation ( purulent hepatitis ) , which may involve the portal vein branches to produce pylephlebitic abscesses , with further throm- botic extension along the portal vein tree . But the pylephlebitic abscesses may develop from ...
Page 102
... abscesses ) which eventually communicate with the intestinal lumen and permit the release of trophozoites . Other trophozoites are excreted in the encysted form , maintain- ing the life cycle . The described events may proceed ...
... abscesses ) which eventually communicate with the intestinal lumen and permit the release of trophozoites . Other trophozoites are excreted in the encysted form , maintain- ing the life cycle . The described events may proceed ...
Page 103
... abscesses develop ; even endocarditis has been reported . The liver is relatively rarely the site of actinomycotic abscesses . In the major- ity of such instances , the primary focus is in the proximal colon , especially in the appendix ...
... abscesses develop ; even endocarditis has been reported . The liver is relatively rarely the site of actinomycotic abscesses . In the major- ity of such instances , the primary focus is in the proximal colon , especially in the appendix ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall