The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 43
... activity is elevated with increased osteo- blastic activity . It is very high in such bone diseases as ricketts , osteomalacia and Paget's disease . It is moderately elevated with most carcinoma metastases to the bone , especially so if ...
... activity is elevated with increased osteo- blastic activity . It is very high in such bone diseases as ricketts , osteomalacia and Paget's disease . It is moderately elevated with most carcinoma metastases to the bone , especially so if ...
Page 55
... activity but not the volume of the secretion . Vagotomy and parasympatho- lytic drugs ( atropine ) conversely decrease enzymatic activity . The volume of the pancreatic secretion is raised by the injec- tion of secretin , a hormone now ...
... activity but not the volume of the secretion . Vagotomy and parasympatho- lytic drugs ( atropine ) conversely decrease enzymatic activity . The volume of the pancreatic secretion is raised by the injec- tion of secretin , a hormone now ...
Page 58
... activity is expressed in units or milligrams of glucose liberated by 100 ml . of serum . The normal values vary greatly in the literature . Widely accepted is 105 units , with a standard deviation of 26 units , as reported in a study of ...
... activity is expressed in units or milligrams of glucose liberated by 100 ml . of serum . The normal values vary greatly in the literature . Widely accepted is 105 units , with a standard deviation of 26 units , as reported in a study of ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
2 other sections not shown
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall