The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 90
... alterations of the liver may be induced when the hepatic blood flow or oxygen supply suffers secondarily to disturbances elsewhere in the organism . A typical example is shock , which might induce hepatic hyperemia with focal ...
... alterations of the liver may be induced when the hepatic blood flow or oxygen supply suffers secondarily to disturbances elsewhere in the organism . A typical example is shock , which might induce hepatic hyperemia with focal ...
Page 96
... alterations frequently persist with no , little or conspicuous jaundice . In most cases these residual changes are relatively mild and may disappear within 1 year . The incidence of permanent disease seems to be rather small . The ...
... alterations frequently persist with no , little or conspicuous jaundice . In most cases these residual changes are relatively mild and may disappear within 1 year . The incidence of permanent disease seems to be rather small . The ...
Page 117
... alterations stand in the foreground of autopsy findings in at least one half of the cases . Irregularly shaped ... altered significantly . Biopsy specimens obtained . from nonfatal cases of eclampsia have re- vealed similar but more ...
... alterations stand in the foreground of autopsy findings in at least one half of the cases . Irregularly shaped ... altered significantly . Biopsy specimens obtained . from nonfatal cases of eclampsia have re- vealed similar but more ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall