The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 83
... appear nor- mal . At this stage hyperbilirubinemia and bilirubinuria appear , the activity of serum alkaline phosphatase is elevated and the urinary urobilinogen excretion is reduced . Subsequently , cellular infiltration of the portal ...
... appear nor- mal . At this stage hyperbilirubinemia and bilirubinuria appear , the activity of serum alkaline phosphatase is elevated and the urinary urobilinogen excretion is reduced . Subsequently , cellular infiltration of the portal ...
Page 94
... appears in this fulminant form in civil- ians ( including children ) , as well as in military personnel . But it should ... appear normal , because within a period of a few days the serum does not yet reflect the metabolic disturbance ...
... appears in this fulminant form in civil- ians ( including children ) , as well as in military personnel . But it should ... appear normal , because within a period of a few days the serum does not yet reflect the metabolic disturbance ...
Page 124
... appear well established . One is an increased concentration of one of the crucial substances in the bile . Although the constitution of the bile does not neces- sarily reflect that of the blood serum , elevation of either serum ...
... appear well established . One is an increased concentration of one of the crucial substances in the bile . Although the constitution of the bile does not neces- sarily reflect that of the blood serum , elevation of either serum ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall