The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 7
Neutral fat is found in the form of droplets , which are stainable in frozen sections
by fat stains but appear as vacuoles after dissolution of fat with the routine use of
organic solvents in histologic techniques . The fat droplets or vacuoles in normal
...
Neutral fat is found in the form of droplets , which are stainable in frozen sections
by fat stains but appear as vacuoles after dissolution of fat with the routine use of
organic solvents in histologic techniques . The fat droplets or vacuoles in normal
...
Page 83
The change first to appear is the accumulation of bile pigment in the liver cells
and Kupffer cells in the central zone of the lobule . Simultaneously , bile may
amass in the form of ramified bile plugs in the dilated bile capillaries . The
cytoplasm of ...
The change first to appear is the accumulation of bile pigment in the liver cells
and Kupffer cells in the central zone of the lobule . Simultaneously , bile may
amass in the form of ramified bile plugs in the dilated bile capillaries . The
cytoplasm of ...
Page 124
The causes of cholelithiasis , however , appear well established . One is an
increased concentration of one of the crucial substances in the bile . Although the
constitution of the bile does not necessarily reflect that of the blood serum ,
elevation ...
The causes of cholelithiasis , however , appear well established . One is an
increased concentration of one of the crucial substances in the bile . Although the
constitution of the bile does not necessarily reflect that of the blood serum ,
elevation ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
96 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall