The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 62
... associated macroscopically with an enlarged organ , a tense capsule , a rounded anterior edge , reduced consistency and an obscured architecture , provided that passive con- gestion does not accentuate it . A brown- ish - red hue ...
... associated macroscopically with an enlarged organ , a tense capsule , a rounded anterior edge , reduced consistency and an obscured architecture , provided that passive con- gestion does not accentuate it . A brown- ish - red hue ...
Page 90
... associated with a reduced volume of circulating blood ; ( 2 ) oxygen lack , especially in hemorrhagic shock ; ( 3 ) impeded hepatic circulation , essen- tially by a throttle mechanism of the hepatic veins , leading to centrolobular ...
... associated with a reduced volume of circulating blood ; ( 2 ) oxygen lack , especially in hemorrhagic shock ; ( 3 ) impeded hepatic circulation , essen- tially by a throttle mechanism of the hepatic veins , leading to centrolobular ...
Page 123
... associated with abnormalities of the bile duct system which are of greater clinical significance . Con- genital hypoplasia , valve formation or angulation of the common bile duct may be the cause of a pro- tracted partial obstruction ...
... associated with abnormalities of the bile duct system which are of greater clinical significance . Con- genital hypoplasia , valve formation or angulation of the common bile duct may be the cause of a pro- tracted partial obstruction ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall