The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 47
The resulting biliverdin-iron- globin product, called verdohemoglobin, loses its
iron and globin components and becomes biliverdin, which, subsequentlv, is
transformed by reduction into bilirubin. This pigment gives the red diazo reaction
(with ...
The resulting biliverdin-iron- globin product, called verdohemoglobin, loses its
iron and globin components and becomes biliverdin, which, subsequentlv, is
transformed by reduction into bilirubin. This pigment gives the red diazo reaction
(with ...
Page 48
JAUNDICE WITHOUT IMPAIRMENT OF BILE FLOW OVERPRODUCTION' -
RETENTION HEMOLYTIC BLOOD STREAM KEY BILIVERDINGLOBIN
INDIRECT BILIRUBIN X PROMPT REACTING BILIRUBIN UROBILINOGEN
FAMILIAL BLOOD ...
JAUNDICE WITHOUT IMPAIRMENT OF BILE FLOW OVERPRODUCTION' -
RETENTION HEMOLYTIC BLOOD STREAM KEY BILIVERDINGLOBIN
INDIRECT BILIRUBIN X PROMPT REACTING BILIRUBIN UROBILINOGEN
FAMILIAL BLOOD ...
Page 124
Although the constitution of the bile does not necessarilv reflect that of the blood
serum, elevation of either serum cholesterol or bilirubin may result in their
increase in the bile, with their subsequent precipitation giving rise to stone
formation.
Although the constitution of the bile does not necessarilv reflect that of the blood
serum, elevation of either serum cholesterol or bilirubin may result in their
increase in the bile, with their subsequent precipitation giving rise to stone
formation.
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Contents
DISEASES OF THE LIVER continued | 3 |
PLATE PAGE NUMBER NUMBER 24 CarbohydratcLipid Disturbances | 86 |
Wilsons Disease | 87 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis alkaline phosphatase Amer anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholestasis cholesterol chronic cirrhosis clinical common bile duct connective tissue cystic duct cysts damage degeneration develop diagnosis dilated duodenal duodenum enlarged excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular histologic infection inferior intestinal intrahepatic islet jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobule lymph lymphatic metabolism metastases mucosa necrosis necrotic nodes normal onlv organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior primary hepatic protein rare regenerative nodules result septa serum sinusoids sometimes sphincter spleen splenic stage stones superior mesenteric surface surgical tion tumor urobilinogen usuallv usually vascular vessels viral hepatitis wall