The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 56
GENERAL CIRCULATION AMYLASE LIPASE DIGESTION BY TRYPSIN
BIOCHEMICAL CHANGES IN Acute PANCREATITIS CALCIUM IL BLOOD
CALCIUM REDUCED > FAT NECROSIS The most characteristic changes in
acute pancreatitis ...
GENERAL CIRCULATION AMYLASE LIPASE DIGESTION BY TRYPSIN
BIOCHEMICAL CHANGES IN Acute PANCREATITIS CALCIUM IL BLOOD
CALCIUM REDUCED > FAT NECROSIS The most characteristic changes in
acute pancreatitis ...
Page 75
PERSONALITY CHANGES , VACANT STARE STAGE II LETHARGY , FLAPPING
TREMOR , MUSCLE TWITCHING HEPATIC COMA 20 STAGE III NOISY ,
ABUSIVE , VIOLENT FOETOR HEPATICUS KNEE CLONUS ANKLE CLONUS +
...
PERSONALITY CHANGES , VACANT STARE STAGE II LETHARGY , FLAPPING
TREMOR , MUSCLE TWITCHING HEPATIC COMA 20 STAGE III NOISY ,
ABUSIVE , VIOLENT FOETOR HEPATICUS KNEE CLONUS ANKLE CLONUS +
...
Page 163
... 40 mental changes , 75 , 86 , 87 , 119 , 120 mercapturic acid , 44 , 64
mesentery , see peritoneum mesobilirubinogen ... 82 , 125 , 126 , 127 , 132 , 134
infants , 119 ( versus ) intrahepatic obstruction , 82 , 83 liver changes , 43 , 49 , 51
, 83 ...
... 40 mental changes , 75 , 86 , 87 , 119 , 120 mercapturic acid , 44 , 64
mesentery , see peritoneum mesobilirubinogen ... 82 , 125 , 126 , 127 , 132 , 134
infants , 119 ( versus ) intrahepatic obstruction , 82 , 83 liver changes , 43 , 49 , 51
, 83 ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall