The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 11
... cholangioles or independently perforate the limiting plate . The intralobular cholangioles are surrounded by a connective tissue sheet which also envelops the arterioles and possibly very small lymphatic vessels . The epithelial cells ...
... cholangioles or independently perforate the limiting plate . The intralobular cholangioles are surrounded by a connective tissue sheet which also envelops the arterioles and possibly very small lymphatic vessels . The epithelial cells ...
Page 83
... cholangioles becomes more marked , and bile casts form even in peripheral bile capillaries . The dilated cholangioles contain thick bile plugs , microcalculi , especially on the border between the lobular parenchyma and the connective ...
... cholangioles becomes more marked , and bile casts form even in peripheral bile capillaries . The dilated cholangioles contain thick bile plugs , microcalculi , especially on the border between the lobular parenchyma and the connective ...
Page 97
... cholangioles and smallest portal ducts are missing , apparently as a result of destruction by the inflammatory tissue . Fibrosis around proliferated intra- lobular cholangioles obscures the lobular architecture but does not destroy it ...
... cholangioles and smallest portal ducts are missing , apparently as a result of destruction by the inflammatory tissue . Fibrosis around proliferated intra- lobular cholangioles obscures the lobular architecture but does not destroy it ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall