The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 70
... result of liver cell damage , and / or loss of hepatic functions due to diversion of blood by extrahepatic portacaval collaterals and intrahepatic portahepatic venous anastomoses ( see page 69 ) , explain many clinical symptoms .
... result of liver cell damage , and / or loss of hepatic functions due to diversion of blood by extrahepatic portacaval collaterals and intrahepatic portahepatic venous anastomoses ( see page 69 ) , explain many clinical symptoms .
Page 96
In children the disease completely heals shortly after the disappearance of jaundice , whereas in adults clinical , laboratory or anatomic residual alterations frequently persist with no , little or conspicuous jaundice .
In children the disease completely heals shortly after the disappearance of jaundice , whereas in adults clinical , laboratory or anatomic residual alterations frequently persist with no , little or conspicuous jaundice .
Page 123
Most of them have little clinical significance and are encountered as incidental findings on roentgenologic examination , surgical exploration or at autopsy . But any aberration from the norm is a potential cause for stasis in the ...
Most of them have little clinical significance and are encountered as incidental findings on roentgenologic examination , surgical exploration or at autopsy . But any aberration from the norm is a potential cause for stasis in the ...
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall