The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 18
PANCREATICODUODENAL VEIN SPLENIC VEIN INFERIOR MESENTERIC
VEIN MIDDLE COLIC VEIN LEFT COLIC VEIN RIGHT COLIC VEIN ILEOCOLIC
VEIN SUPERIOR HEMORRHOIDAL VEINS The portal vein forms behind the
head of ...
PANCREATICODUODENAL VEIN SPLENIC VEIN INFERIOR MESENTERIC
VEIN MIDDLE COLIC VEIN LEFT COLIC VEIN RIGHT COLIC VEIN ILEOCOLIC
VEIN SUPERIOR HEMORRHOIDAL VEINS The portal vein forms behind the
head of ...
Page 19
PANCREATIC VEINS LEFT GASTRO - EPIPLOIC VEIN - INFERIOR
MESENTERIC VEIN - LEFT COLIC VEIN INFERIOR MESENTERIC VEIN MAY
ENTER JUNCTION OF SPLENIC AND SUPERIOR MESENTERIC R . AND L .
HEPATIC ...
PANCREATIC VEINS LEFT GASTRO - EPIPLOIC VEIN - INFERIOR
MESENTERIC VEIN - LEFT COLIC VEIN INFERIOR MESENTERIC VEIN MAY
ENTER JUNCTION OF SPLENIC AND SUPERIOR MESENTERIC R . AND L .
HEPATIC ...
Page 29
The gastro - epiploic vessels ( see pages 14 and 15 ) may be spared , and the
middle colic vessels must be gently dissected off the pancreas . The transverse
mesocolon is in contact with the anterior surface of the head and passes along
the ...
The gastro - epiploic vessels ( see pages 14 and 15 ) may be spared , and the
middle colic vessels must be gently dissected off the pancreas . The transverse
mesocolon is in contact with the anterior surface of the head and passes along
the ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall