The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 61
... complete or partial situs inversus are readily resolved by roentgenologic examination . The causes of situs inversus have not been estab- lished , and the explanations offered are all hypothet- ical . In complete situs inversus , the ...
... complete or partial situs inversus are readily resolved by roentgenologic examination . The causes of situs inversus have not been estab- lished , and the explanations offered are all hypothet- ical . In complete situs inversus , the ...
Page 82
... complete or incomplete and , in the latter case , it is often intermittent . Complete obstruction is most fre- quently caused by tumors , which initially produce an incomplete and , subse- quently , a permanent complete occlu- sion ...
... complete or incomplete and , in the latter case , it is often intermittent . Complete obstruction is most fre- quently caused by tumors , which initially produce an incomplete and , subse- quently , a permanent complete occlu- sion ...
Page 123
... complete " . Even the complete form does not , as a rule , cause clinical symptoms , in contrast to previously held beliefs . Complete dupli- cation of the gallbladder , with or without two independent cystic ducts , is merely an ...
... complete " . Even the complete form does not , as a rule , cause clinical symptoms , in contrast to previously held beliefs . Complete dupli- cation of the gallbladder , with or without two independent cystic ducts , is merely an ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall