The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 67
... connective tissue framework surrounding the fatty cysts increases , becomes dense and acts almost like a capsule . When eventually the fat disappears from the cysts via adjacent sinusoids or bile canaliculi , the condensed connective ...
... connective tissue framework surrounding the fatty cysts increases , becomes dense and acts almost like a capsule . When eventually the fat disappears from the cysts via adjacent sinusoids or bile canaliculi , the condensed connective ...
Page 68
... connective tissue frame- work collapses . Portal triads and central fields become approximated and a postnecrotic scar de- velops , in which some sinusoids are transformed into venous channels shunting blood from the portal canals to ...
... connective tissue frame- work collapses . Portal triads and central fields become approximated and a postnecrotic scar de- velops , in which some sinusoids are transformed into venous channels shunting blood from the portal canals to ...
Page 111
... connective tissue ( see also page 60 ) . Solid , nodular hamartomas differ in color and architecture from the surround- ing parenchyma . With no specific locali- zation , their cut surface appears round if they are small , and ...
... connective tissue ( see also page 60 ) . Solid , nodular hamartomas differ in color and architecture from the surround- ing parenchyma . With no specific locali- zation , their cut surface appears round if they are small , and ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall