The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 67
The connective tissue framework surrounding the fatty cysts increases , becomes dense and acts almost like a capsule . When eventually the fat disappears from the cysts via adjacent sinusoids or bile canaliculi , the condensed ...
The connective tissue framework surrounding the fatty cysts increases , becomes dense and acts almost like a capsule . When eventually the fat disappears from the cysts via adjacent sinusoids or bile canaliculi , the condensed ...
Page 68
The process starts in the necrotic areas , where the connective tissue framework collapses . Portal triads and central fields become approximated and a postnecrotic scar develops , in which some sinusoids are transformed into venous ...
The process starts in the necrotic areas , where the connective tissue framework collapses . Portal triads and central fields become approximated and a postnecrotic scar develops , in which some sinusoids are transformed into venous ...
Page 111
The smallest hamartomas , called microhamartomas or Meyenberg complexes , are irregular bile duct proliferations surrounded by proliferated connective tissue ( see also page 60 ) . Solid , nodular hamartomas differ in color and ...
The smallest hamartomas , called microhamartomas or Meyenberg complexes , are irregular bile duct proliferations surrounded by proliferated connective tissue ( see also page 60 ) . Solid , nodular hamartomas differ in color and ...
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall