The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 67
The connective tissue framework surrounding the fatty cysts increases , becomes
dense and acts almost like a capsule . When eventually the fat disappears from
the cysts via adjacent sinusoids or bile canaliculi , the condensed connective ...
The connective tissue framework surrounding the fatty cysts increases , becomes
dense and acts almost like a capsule . When eventually the fat disappears from
the cysts via adjacent sinusoids or bile canaliculi , the condensed connective ...
Page 68
The process starts in the necrotic areas , where the connective tissue framework
collapses . Portal triads and central fields become approximated and a
postnecrotic scar develops , in which some sinusoids are transformed into
venous ...
The process starts in the necrotic areas , where the connective tissue framework
collapses . Portal triads and central fields become approximated and a
postnecrotic scar develops , in which some sinusoids are transformed into
venous ...
Page 111
The smallest hamartomas , called microhamartomas or Meyenberg complexes ,
are irregular bile duct proliferations surrounded by proliferated connective tissue
( see also page 60 ) . Solid , nodular hamartomas differ in color and architecture ...
The smallest hamartomas , called microhamartomas or Meyenberg complexes ,
are irregular bile duct proliferations surrounded by proliferated connective tissue
( see also page 60 ) . Solid , nodular hamartomas differ in color and architecture ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
96 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall