The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
From inside the book
Results 1-3 of 3
Page 47
... diarrhea does bilirubin appear in the feces . The main fecal pigment is urobilin , the RETICULO- ENDOTHELIAL CELL RED CELL URINE UROBILINOGEN + BOWEL PORTAL VEIN Netter M.D. OCIBA STOOL intestinal oxidation product of a part of the ...
... diarrhea does bilirubin appear in the feces . The main fecal pigment is urobilin , the RETICULO- ENDOTHELIAL CELL RED CELL URINE UROBILINOGEN + BOWEL PORTAL VEIN Netter M.D. OCIBA STOOL intestinal oxidation product of a part of the ...
Page 132
... diarrhea . Barium ene- mas usually reveal the fistula's tract . The essential symptom of a cholecystogastric fistula is the vomiting of a gallstone , though such an event could also result from duodenal regurgitation . Occasion- ally ...
... diarrhea . Barium ene- mas usually reveal the fistula's tract . The essential symptom of a cholecystogastric fistula is the vomiting of a gallstone , though such an event could also result from duodenal regurgitation . Occasion- ally ...
Page 148
... diarrhea or large , bulky stools con- taining much undigested fat , are fairly characteristic of those patients whose pancreatic duct is obstructed ( see page 57 ) . The pain , which may be colicky or intermittent in type , is much the ...
... diarrhea or large , bulky stools con- taining much undigested fat , are fairly characteristic of those patients whose pancreatic duct is obstructed ( see page 57 ) . The pain , which may be colicky or intermittent in type , is much the ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
2 other sections not shown
Other editions - View all
Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall