The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 11
... dilated and filled with bile , sometimes precipitating to bile casts or plugs which probably contain a core of precipitated protein . Under these cir- cumstances the arrangement of the bile canaliculi is readily visualized without ...
... dilated and filled with bile , sometimes precipitating to bile casts or plugs which probably contain a core of precipitated protein . Under these cir- cumstances the arrangement of the bile canaliculi is readily visualized without ...
Page 69
... dilated but are seen only at operation or autopsy . The veins in the anterior abdominal wall dilate as a result of collaterals developing around the remnants of the fetal circula- tion in the round ligament . They become especially ...
... dilated but are seen only at operation or autopsy . The veins in the anterior abdominal wall dilate as a result of collaterals developing around the remnants of the fetal circula- tion in the round ligament . They become especially ...
Page 83
... dilated bile capillaries . The cyto- plasm of some liver cells adjacent to the bile capillaries is rarefied and pyknosis ( " feathery degeneration " ) may be seen , whereas the liver cells of the intermedi- ate and peripheral zones ...
... dilated bile capillaries . The cyto- plasm of some liver cells adjacent to the bile capillaries is rarefied and pyknosis ( " feathery degeneration " ) may be seen , whereas the liver cells of the intermedi- ate and peripheral zones ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall