The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 38
... elevated in hepatic disorders of any type , but especially in chronic hepatitis and cirrhosis . The highest values are observed in postnecrotic cirrhosis . In uncomplicated biliary obstruction the elevation is slight but becomes more ...
... elevated in hepatic disorders of any type , but especially in chronic hepatitis and cirrhosis . The highest values are observed in postnecrotic cirrhosis . In uncomplicated biliary obstruction the elevation is slight but becomes more ...
Page 43
... elevated with increased osteo- blastic activity . It is very high in such bone diseases as ricketts , osteomalacia and Paget's disease . It is moderately elevated with most carcinoma metastases to the bone , especially so if they are ...
... elevated with increased osteo- blastic activity . It is very high in such bone diseases as ricketts , osteomalacia and Paget's disease . It is moderately elevated with most carcinoma metastases to the bone , especially so if they are ...
Page 56
Frank Henry Netter. SERUM AMYLASE ELEVATED SERUM LIPASE ELEVATED ( CHRONIC PANCREATITIS ) I REDUCTION OF LIBERATED GLUCOSE BY. GENERAL CIRCULATION BIOCHEMICAL CHANGES IN ACUTE PANCREATITIS AMYLASE LIPASE DIGESTION BY TRYPSIN CALCIUM The ...
Frank Henry Netter. SERUM AMYLASE ELEVATED SERUM LIPASE ELEVATED ( CHRONIC PANCREATITIS ) I REDUCTION OF LIBERATED GLUCOSE BY. GENERAL CIRCULATION BIOCHEMICAL CHANGES IN ACUTE PANCREATITIS AMYLASE LIPASE DIGESTION BY TRYPSIN CALCIUM The ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall