The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 43
... EXCRETION IN BILE NORMAL BLOOD LEVEL BLOOD LEVEL HIGH LIVER CELL DAMAGE MARKED ELEVATION OF BLOOD LEVEL BLOOD LEVEL MODERATELY ELEVATED Phosphatases are esterases which hy- drolize reversibly monophosphoric acid esters . These enzymes ...
... EXCRETION IN BILE NORMAL BLOOD LEVEL BLOOD LEVEL HIGH LIVER CELL DAMAGE MARKED ELEVATION OF BLOOD LEVEL BLOOD LEVEL MODERATELY ELEVATED Phosphatases are esterases which hy- drolize reversibly monophosphoric acid esters . These enzymes ...
Page 44
... excreted in the urine . Hippuric acid formation takes EXCRETION OF HIPPURIC ACID IN URINE LIVER DAMAGE DEFICIENCY OF GLYCINE OR DEFICIENCY OF ENZYME INGESTION OF SODIUM BENZOATE place almost entirely in the liver and only to a slight ...
... excreted in the urine . Hippuric acid formation takes EXCRETION OF HIPPURIC ACID IN URINE LIVER DAMAGE DEFICIENCY OF GLYCINE OR DEFICIENCY OF ENZYME INGESTION OF SODIUM BENZOATE place almost entirely in the liver and only to a slight ...
Page 45
... EXCRETION TEST EXCRETION IN BILE INJECTION OF DYE BROMSULPHALEIN TEST ( OBSTRUCTION ; JAUNDICE ) BLOOD DRAWN AFTER 45 MINUTES ALKALI ADDED TO SERUM BROMSULPHALEIN TEST ( LIVER DAMAGE JAUNDICE ) BLOCK BILE CAPILLARY STASIS BLOCK DAMAGED ...
... EXCRETION TEST EXCRETION IN BILE INJECTION OF DYE BROMSULPHALEIN TEST ( OBSTRUCTION ; JAUNDICE ) BLOOD DRAWN AFTER 45 MINUTES ALKALI ADDED TO SERUM BROMSULPHALEIN TEST ( LIVER DAMAGE JAUNDICE ) BLOCK BILE CAPILLARY STASIS BLOCK DAMAGED ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall