The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 40
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
Page 77
... FACTORS Starvation , Malnutrition Hepatic injury can be caused by three kinds of dietary deficiencies : ( 1 ) gen- eral caloric undernutrition ( starvation ) , ( 2 ) lack of one specific dietary compo- nent , and ( 3 ) imbalanced ...
... FACTORS Starvation , Malnutrition Hepatic injury can be caused by three kinds of dietary deficiencies : ( 1 ) gen- eral caloric undernutrition ( starvation ) , ( 2 ) lack of one specific dietary compo- nent , and ( 3 ) imbalanced ...
Page 78
... FACTORS FATTY LIVER LIPOTROPIC FACTORS & Netter M.D. OCIBA the abnormalities of the hepatic test become more significant . Patients with fatty liver seem to be more susceptible to infections ; lobular pneumonia espe cially may take a ...
... FACTORS FATTY LIVER LIPOTROPIC FACTORS & Netter M.D. OCIBA the abnormalities of the hepatic test become more significant . Patients with fatty liver seem to be more susceptible to infections ; lobular pneumonia espe cially may take a ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall