The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 40
The liver produces , furthermore , a number of other factors necessary for the
conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin
or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) .
The liver produces , furthermore , a number of other factors necessary for the
conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin
or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) .
Page 77
LIPOGENIC FACTORS IMPACTED STONE WITH EDEMA COMMON DUCT
OBSTRUCTION : JAUNDICE OBSTRUCTIVE. ALCOHOL INCREASED
METABOLIC NEEDS DECREASED METABOLIC NEEDS SUGAR LIPOTROPIC
FACTORS ...
LIPOGENIC FACTORS IMPACTED STONE WITH EDEMA COMMON DUCT
OBSTRUCTION : JAUNDICE OBSTRUCTIVE. ALCOHOL INCREASED
METABOLIC NEEDS DECREASED METABOLIC NEEDS SUGAR LIPOTROPIC
FACTORS ...
Page 78
( Continued from page 77 ) ALCOHOLISM of lipotropic factors is reduced
absolutely or relatively to the intake of lipogenic factors . Such imbalance , when
persistent , leads invariably to metabolic disturbance in the liver and to fat
accumulation .
( Continued from page 77 ) ALCOHOLISM of lipotropic factors is reduced
absolutely or relatively to the intake of lipogenic factors . Such imbalance , when
persistent , leads invariably to metabolic disturbance in the liver and to fat
accumulation .
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall