The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 54
FAILURE OF ABSORPTION 2. FAILURE OF EXCRETION BY LIVER 3. BILE DUCT OBSTRUCTION WITH BACK PRESSURE ARRESTING SECRETION 4. CYSTIC DUCT OBSTRUCTION 5. FAILURE OF GALLBLADDER ( DISEASED ) TO CONCENTRATE CHOLECYSTOGRAPHY , CHOLANGIOGRAPHY ...
FAILURE OF ABSORPTION 2. FAILURE OF EXCRETION BY LIVER 3. BILE DUCT OBSTRUCTION WITH BACK PRESSURE ARRESTING SECRETION 4. CYSTIC DUCT OBSTRUCTION 5. FAILURE OF GALLBLADDER ( DISEASED ) TO CONCENTRATE CHOLECYSTOGRAPHY , CHOLANGIOGRAPHY ...
Page 74
PORTAL HYPERTENSION 3 HEART FAILURE ASCITES Pathogenesis 2 LIVER DAMAGE A - HYPOPROTEINEMIA ( DECREASED ALBUMIN FORMATION ) B - POSSIBLY DECREASED INACTIVATION OF ANTIDIURETIC HORMONE . HYPOTHETICALLY ALSO DECREASED INACTIVATION ( OR ...
PORTAL HYPERTENSION 3 HEART FAILURE ASCITES Pathogenesis 2 LIVER DAMAGE A - HYPOPROTEINEMIA ( DECREASED ALBUMIN FORMATION ) B - POSSIBLY DECREASED INACTIVATION OF ANTIDIURETIC HORMONE . HYPOTHETICALLY ALSO DECREASED INACTIVATION ( OR ...
Page 76
HEMATURIA GRANULAR CASTS Renal shutdown , with azotemia and hyperkalemia , occasionally complicates the course of failing liver disease . This has been called the " hepatorenal syndrome ” , a term first used about 20 years ago to ...
HEMATURIA GRANULAR CASTS Renal shutdown , with azotemia and hyperkalemia , occasionally complicates the course of failing liver disease . This has been called the " hepatorenal syndrome ” , a term first used about 20 years ago to ...
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall