The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 81
Esophageal varices are less frequent than in the nutritional type of cirrhosis , but splenomegaly , spider nevi and , in later stages , ascites are relatively regular findings . Distinct nodules may be palpable , in contrast to the ...
Esophageal varices are less frequent than in the nutritional type of cirrhosis , but splenomegaly , spider nevi and , in later stages , ascites are relatively regular findings . Distinct nodules may be palpable , in contrast to the ...
Page 96
The picture in some patients is confusing , because clinical and laboratory findings and histologic alterations as recognized by liver biopsy do not match . Quite frequently , only one of the three factors may point to a persisting ...
The picture in some patients is confusing , because clinical and laboratory findings and histologic alterations as recognized by liver biopsy do not match . Quite frequently , only one of the three factors may point to a persisting ...
Page 143
The laboratory findings have been discussed on pages 56 and 58. Hypocalcemia and hypokalemia are frequent findings with severe pancreatitis and fat necrosis . Diabetes may become evident during the acute phase , and permanent if ...
The laboratory findings have been discussed on pages 56 and 58. Hypocalcemia and hypokalemia are frequent findings with severe pancreatitis and fat necrosis . Diabetes may become evident during the acute phase , and permanent if ...
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall