The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 40
Frank Henry Netter. VITAMIN K IN FOOD 1 . NORMAL PROTHROMBIN
FORMATION AMINO - ACIDURIA MILLON ' S REACTION FOR TYROSINE
PROTEIN IN FOOD PAPER CHROMATOGRAM PROTHROMBIN
PROTHROMBIN FORMED IN ...
Frank Henry Netter. VITAMIN K IN FOOD 1 . NORMAL PROTHROMBIN
FORMATION AMINO - ACIDURIA MILLON ' S REACTION FOR TYROSINE
PROTEIN IN FOOD PAPER CHROMATOGRAM PROTHROMBIN
PROTHROMBIN FORMED IN ...
Page 68
The increasing disturbance of the hepatic circulation in the lobular and nodular
parenchyma is also a stimulus for central necrosis as well as peripheral
inflammation , both of which , in turn , provoke septa formation and separation of
single or ...
The increasing disturbance of the hepatic circulation in the lobular and nodular
parenchyma is also a stimulus for central necrosis as well as peripheral
inflammation , both of which , in turn , provoke septa formation and separation of
single or ...
Page 74
PORTAL HYPERTENSION 3 20 HEART FAILURE ASCITES Pathogenesis
LIVER DAMAGE A - HYPOPROTEINEMIA ( DECREASED ALBUMIN
FORMATION ) B - POSSIBLY DECREASEDINACTIVATION OF ANTIDIURETIC
HORMONE .
PORTAL HYPERTENSION 3 20 HEART FAILURE ASCITES Pathogenesis
LIVER DAMAGE A - HYPOPROTEINEMIA ( DECREASED ALBUMIN
FORMATION ) B - POSSIBLY DECREASEDINACTIVATION OF ANTIDIURETIC
HORMONE .
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall