The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 10
... liver can be seen ( " Zuckergussleber " or chronic perihepatitis ) . At the hilus of the liver ( and also in the neighbor- hood of the inferior vena cava ) , Glis- son's capsule thickens and extends along the branches of the portal vein , ...
... liver can be seen ( " Zuckergussleber " or chronic perihepatitis ) . At the hilus of the liver ( and also in the neighbor- hood of the inferior vena cava ) , Glis- son's capsule thickens and extends along the branches of the portal vein , ...
Page 69
... hepatic arteries and portal veins but act rather as short anastomoses between the two vascular systems of the liver . When , in the course of the fibrotic process , the smaller branches of the portal vein become compressed and in part ...
... hepatic arteries and portal veins but act rather as short anastomoses between the two vascular systems of the liver . When , in the course of the fibrotic process , the smaller branches of the portal vein become compressed and in part ...
Page 72
... INTRAHEPATIC CAUSES The portal venous pressure rises above the norm of approximately 20 cm . of water because of ( 1 ) block in the intra- hepatic portal vein tree , ( 2 ) impaired outflow of blood from the liver , ( 3 ) exces- sive ...
... INTRAHEPATIC CAUSES The portal venous pressure rises above the norm of approximately 20 cm . of water because of ( 1 ) block in the intra- hepatic portal vein tree , ( 2 ) impaired outflow of blood from the liver , ( 3 ) exces- sive ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall