The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 84
... infectious cir- rhosis of Mallory , results from biliary obstruction complicated by bacterial infection , which develops from prolonged obstruction by calculi or from strictures of the extrahepatic ducts ( see page 133 ) . This type of ...
... infectious cir- rhosis of Mallory , results from biliary obstruction complicated by bacterial infection , which develops from prolonged obstruction by calculi or from strictures of the extrahepatic ducts ( see page 133 ) . This type of ...
Page 92
... infections the cellular exudate extends in strand form into the lobular parenchyma . Another route of infection , which , contrary to a frequently encountered belief , is relatively rare , follows the bile duct system and occurs mainly ...
... infections the cellular exudate extends in strand form into the lobular parenchyma . Another route of infection , which , contrary to a frequently encountered belief , is relatively rare , follows the bile duct system and occurs mainly ...
Page 127
... infection , bile pigment and bile acids are gradually reabsorbed . Since new bile cannot enter and the mucous secretion cannot leave the gallbladder , the enlarged organ becomes filled with a white and mucous fluid ; this prompted the ...
... infection , bile pigment and bile acids are gradually reabsorbed . Since new bile cannot enter and the mucous secretion cannot leave the gallbladder , the enlarged organ becomes filled with a white and mucous fluid ; this prompted the ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
2 other sections not shown
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall