The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 91
Toxic INJURIES CARBON TETRACHLORIDE POISONING A great variety of
clinical manifestations may result from liver injury by toxic substances which may
belong to some notwell - defined endogenous factors ( breakdown products of ...
Toxic INJURIES CARBON TETRACHLORIDE POISONING A great variety of
clinical manifestations may result from liver injury by toxic substances which may
belong to some notwell - defined endogenous factors ( breakdown products of ...
Page 133
A too rapid appliance of hemostats may result in a crushing injury of a yet
incompletely dissected common bile duct . A retrograde dissection of the
gallbladder fundus and displacement of the gallbladder to the left side of the
abdomen may ...
A too rapid appliance of hemostats may result in a crushing injury of a yet
incompletely dissected common bile duct . A retrograde dissection of the
gallbladder fundus and displacement of the gallbladder to the left side of the
abdomen may ...
Page 162
... 51 , 92 diffuse degeneration , 62 duct injuries ( following ) , 133 duodenal
drainage , 53 eclampsia , 117 eosinophilic ... 77 , 78 portal hypertension , 72
pregnancy , 117 rupture tendency , 116 toxic injury , 62 , 78 , 91 yellow fever , 98
fibrin ...
... 51 , 92 diffuse degeneration , 62 duct injuries ( following ) , 133 duodenal
drainage , 53 eclampsia , 117 eosinophilic ... 77 , 78 portal hypertension , 72
pregnancy , 117 rupture tendency , 116 toxic injury , 62 , 78 , 91 yellow fever , 98
fibrin ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall