The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 41
Provided intestinal absorption is not increased as in hyperthyroidism or
decreased as in deficiency diseases , and provided renal insufficiency does not
interfere , a large amount of galactose in the urine may be considered an
indication of liver ...
Provided intestinal absorption is not increased as in hyperthyroidism or
decreased as in deficiency diseases , and provided renal insufficiency does not
interfere , a large amount of galactose in the urine may be considered an
indication of liver ...
Page 88
Except in hypo - or achlorhydria only the ferrous form enters the intestine and
passes through the mucosal membrane . During this ... When ferritin in the
intestinal wall releases its iron to the circulating blood , it is reverted back to
apoferritin .
Except in hypo - or achlorhydria only the ferrous form enters the intestine and
passes through the mucosal membrane . During this ... When ferritin in the
intestinal wall releases its iron to the circulating blood , it is reverted back to
apoferritin .
Page 102
INTESTINAL FOCUS 3 . ... Vetlar M . D . ( OCIBA Amebiasis , transmitted by the
protozoan parasite Endamoeba histolytica , is initially and predominantly an
intestinal disease , but it involves the liver in one tenth to one third of the cases .
INTESTINAL FOCUS 3 . ... Vetlar M . D . ( OCIBA Amebiasis , transmitted by the
protozoan parasite Endamoeba histolytica , is initially and predominantly an
intestinal disease , but it involves the liver in one tenth to one third of the cases .
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall