The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 55
... larger volume the total amount of these ions is still greater than without secretin ... LARGE BOWEL TRYPSINOGEN DEXTRINS STARCH AMYLASE FAT LIPASE PROTEIN TRYPSIN ... intestine , appear in the accompanying diagrammatic picture . When fat ...
... larger volume the total amount of these ions is still greater than without secretin ... LARGE BOWEL TRYPSINOGEN DEXTRINS STARCH AMYLASE FAT LIPASE PROTEIN TRYPSIN ... intestine , appear in the accompanying diagrammatic picture . When fat ...
Page 92
... intestinal wall may be responsible for the permeation of bacteria , so that the portal vein becomes the most important ... large and small intestine , and in newborn children by omphalic infections ( see page 118 ) . But even before the ...
... intestinal wall may be responsible for the permeation of bacteria , so that the portal vein becomes the most important ... large and small intestine , and in newborn children by omphalic infections ( see page 118 ) . But even before the ...
Page 132
... large to pass the cystic duct ( see page 127 ) and a subsequent ... bowel obstruction by stones is less likely than severe biliary tract infections with ... INTESTINE ; GALLBLADDER CONTRACTED SURGICAL END TO END FISTULA ( " ROUX EN Y ...
... large to pass the cystic duct ( see page 127 ) and a subsequent ... bowel obstruction by stones is less likely than severe biliary tract infections with ... INTESTINE ; GALLBLADDER CONTRACTED SURGICAL END TO END FISTULA ( " ROUX EN Y ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall