The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 60
... lead to an irregular arrange- ment of the ducts , resulting in solid nodules or in cysts . Small irregular pro ... leads to cyst formation when the hamar- tomatous cavities become large or com- municate with each other . The large ones ...
... lead to an irregular arrange- ment of the ducts , resulting in solid nodules or in cysts . Small irregular pro ... leads to cyst formation when the hamar- tomatous cavities become large or com- municate with each other . The large ones ...
Page 86
... leads to retardation of development and growth , marked hepatomegaly ( without jaundice ) and splenomegaly . The chil- dren succumb easily to infections . Only few have lived into adolescence . Glucose- tolerance curves show a diabetic ...
... leads to retardation of development and growth , marked hepatomegaly ( without jaundice ) and splenomegaly . The chil- dren succumb easily to infections . Only few have lived into adolescence . Glucose- tolerance curves show a diabetic ...
Page 116
... lead to ruptures or lacerations varying in size and sometimes in num- ber . They are most commonly the result of ... leads to rapid ischemic necro- sis , which , if the patient survives , may be surrounded by a demarcation zone with ...
... lead to ruptures or lacerations varying in size and sometimes in num- ber . They are most commonly the result of ... leads to rapid ischemic necro- sis , which , if the patient survives , may be surrounded by a demarcation zone with ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall