The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 48
JAUNDICE WITHOUT IMPAIRMENT OF BILE FLOW OVERPRODUCTION
RETENTION HEMOLYTIC FAMILIAL BILE CANALICULI BILE CANALICULI
BLOOD STREAM BLOOD STREAM van den BERGH PROMPT O ( LESS THAN 0
. 2 mg .
JAUNDICE WITHOUT IMPAIRMENT OF BILE FLOW OVERPRODUCTION
RETENTION HEMOLYTIC FAMILIAL BILE CANALICULI BILE CANALICULI
BLOOD STREAM BLOOD STREAM van den BERGH PROMPT O ( LESS THAN 0
. 2 mg .
Page 84
Regenerative nodules are rarer , compression of hepatic vein branches is less
significant , and anastomoses between hepatic artery and venous branches are
less developed than in other types of cirrhosis and , consequently , portal ...
Regenerative nodules are rarer , compression of hepatic vein branches is less
significant , and anastomoses between hepatic artery and venous branches are
less developed than in other types of cirrhosis and , consequently , portal ...
Page 134
In contrast , with obstruction by a malignant tumor , the incidence of biliary colic is
far less ( about 25 per cent ) . A more continuous type of pain , with complaints of
localized pressure , fullness or discomfort , though localized in the upper ...
In contrast , with obstruction by a malignant tumor , the incidence of biliary colic is
far less ( about 25 per cent ) . A more continuous type of pain , with complaints of
localized pressure , fullness or discomfort , though localized in the upper ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall