The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 22
The organ possesses no muscular fibers in the mucosa and no submucosal layer
. The fibers of the gallbladder muscle below the mucosa are discontinuous ,
separated by connective tissue and course longitudinally in the inner and ...
The organ possesses no muscular fibers in the mucosa and no submucosal layer
. The fibers of the gallbladder muscle below the mucosa are discontinuous ,
separated by connective tissue and course longitudinally in the inner and ...
Page 89
The bone marrow and the lymph nodes are surprisingly little affected , but the
mucosal membranes , particularly the gastric mucosa , are heavily pigmented .
Many theories to explain the etiology and pathogenesis of hemochromatosis
have ...
The bone marrow and the lymph nodes are surprisingly little affected , but the
mucosal membranes , particularly the gastric mucosa , are heavily pigmented .
Many theories to explain the etiology and pathogenesis of hemochromatosis
have ...
Page 129
From the reddish - green mucosa many irregular - shaped yellow specks
protrude , remindful of the seeds of a ... These histiocytes accumulate in groups
within the mucosal folds of the gallbladder , which lose their regularity and
normal shape ...
From the reddish - green mucosa many irregular - shaped yellow specks
protrude , remindful of the seeds of a ... These histiocytes accumulate in groups
within the mucosal folds of the gallbladder , which lose their regularity and
normal shape ...
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Contents
SECTION XV | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
17 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice Kupffer cells latter lead lesions less liver cells lobe lobular manifestations necrosis nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment plates portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones structures superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall