The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 64
Necrosis implies not only death of cells but also the phenomena following cell
death, namely, the disappearance of cells and frequently also the environmental
reactions to and the vanishing of dead cells. The final and irreversible stage of ...
Necrosis implies not only death of cells but also the phenomena following cell
death, namely, the disappearance of cells and frequently also the environmental
reactions to and the vanishing of dead cells. The final and irreversible stage of ...
Page 68
The increasing disturbance of the hepatic circulation in the lobular and nodular
parenchyma is also a stimulus for central necrosis as well as peripheral
inflammation, both of which, in turn, provoke septa formation and separation of
single or ...
The increasing disturbance of the hepatic circulation in the lobular and nodular
parenchyma is also a stimulus for central necrosis as well as peripheral
inflammation, both of which, in turn, provoke septa formation and separation of
single or ...
Page 95
Subacute Fatal Form If massive necrosis, instead of seizing more or less
simultaneously all lobules, as in acute fulminant hepatitis (sec page 94), is
restricted to only some lobules at one time but proceeds at intervals from one part
of the liver to ...
Subacute Fatal Form If massive necrosis, instead of seizing more or less
simultaneously all lobules, as in acute fulminant hepatitis (sec page 94), is
restricted to only some lobules at one time but proceeds at intervals from one part
of the liver to ...
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Contents
DISEASES OF THE LIVER continued | 3 |
PLATE PAGE NUMBER NUMBER 24 CarbohydratcLipid Disturbances | 86 |
Wilsons Disease | 87 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis alkaline phosphatase Amer anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholestasis cholesterol chronic cirrhosis clinical common bile duct connective tissue cystic duct cysts damage degeneration develop diagnosis dilated duodenal duodenum enlarged excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular histologic infection inferior intestinal intrahepatic islet jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobule lymph lymphatic metabolism metastases mucosa necrosis necrotic nodes normal onlv organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior primary hepatic protein rare regenerative nodules result septa serum sinusoids sometimes sphincter spleen splenic stage stones superior mesenteric surface surgical tion tumor urobilinogen usuallv usually vascular vessels viral hepatitis wall