The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 64
Frank Henry Netter. SECTION XVII - PLATE 5 HEPATIC NECROSIS Necrosis implies not only death of cells but also the phenomena following ... NECROSIS WITH BRIDGING 6 MASSIVE NECROSIS MASSIVE 64 Surgical Approaches to Pancreas Hepatic Necrosis.
Frank Henry Netter. SECTION XVII - PLATE 5 HEPATIC NECROSIS Necrosis implies not only death of cells but also the phenomena following ... NECROSIS WITH BRIDGING 6 MASSIVE NECROSIS MASSIVE 64 Surgical Approaches to Pancreas Hepatic Necrosis.
Page 95
... necrosis , instead of seizing . more or less simultaneously all lobules , as in acute fulminant hepatitis ( see page 94 ) , is restricted to only some lobules at one time but proceeds at intervals from one part of the liver to another ...
... necrosis , instead of seizing . more or less simultaneously all lobules , as in acute fulminant hepatitis ( see page 94 ) , is restricted to only some lobules at one time but proceeds at intervals from one part of the liver to another ...
Page 143
... necrosis . Diabetes may become evident during the acute phase , and permanent if patients with necrosis of the gland survive . Edema occluding the common bile duct or , in later phases , scar tissue may cause jaun- dice . Splenic vein ...
... necrosis . Diabetes may become evident during the acute phase , and permanent if patients with necrosis of the gland survive . Edema occluding the common bile duct or , in later phases , scar tissue may cause jaun- dice . Splenic vein ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall