The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
From inside the book
Results 1-3 of 41
Page 64
SECTION XVII - PLATE 5 HEPATIC NECROSIS Necrosis implies not only death
of cells but also the phenomena following cell death , namely , the
disappearance of cells and frequently also the environmental reactions to and
the vanishing of ...
SECTION XVII - PLATE 5 HEPATIC NECROSIS Necrosis implies not only death
of cells but also the phenomena following cell death , namely , the
disappearance of cells and frequently also the environmental reactions to and
the vanishing of ...
Page 65
BA SUBMASSIVE COLLAPSE HEPATIC TISSUE COLLAPSES AFTER MASSIVE
OR SUBMASSIVE NECROSIS OF AN. • FOCAL NECROSIS CENTRAL
NECROSIS PERIPORTAL PERIPHERAL ) ONECROSIS O CENTRAL
NECROSIS 9 ...
BA SUBMASSIVE COLLAPSE HEPATIC TISSUE COLLAPSES AFTER MASSIVE
OR SUBMASSIVE NECROSIS OF AN. • FOCAL NECROSIS CENTRAL
NECROSIS PERIPORTAL PERIPHERAL ) ONECROSIS O CENTRAL
NECROSIS 9 ...
Page 95
The duration of such a process varies from several weeks to many months ,
depending upon the speed of necrosis and the acuity of the individual bouts .
Similarly as with fulminant hepatitis , the etiology of the subacute fatal hepatitis is
not ...
The duration of such a process varies from several weeks to many months ,
depending upon the speed of necrosis and the acuity of the individual bouts .
Similarly as with fulminant hepatitis , the etiology of the subacute fatal hepatitis is
not ...
What people are saying - Write a review
We haven't found any reviews in the usual places.
Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
96 other sections not shown
Other editions - View all
Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall