The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 68
... nodular paren- chyma is also a stimulus for central necrosis as well as peripheral inflammation , both of which , in turn , provoke septa formation and separation of single or small groups of cells and , therewith , regenerative nodules ...
... nodular paren- chyma is also a stimulus for central necrosis as well as peripheral inflammation , both of which , in turn , provoke septa formation and separation of single or small groups of cells and , therewith , regenerative nodules ...
Page 81
... nodular liver is the most fre- quent picture encountered at autopsy . NODULES WITH RELATIVELY NORMAL LOBULAR ARCHITECTURE AND REGENERATIVE NODULES SEPARATED BY THICK FIBROUS BANDS Its characteristic features are an uneven distribution of ...
... nodular liver is the most fre- quent picture encountered at autopsy . NODULES WITH RELATIVELY NORMAL LOBULAR ARCHITECTURE AND REGENERATIVE NODULES SEPARATED BY THICK FIBROUS BANDS Its characteristic features are an uneven distribution of ...
Page 111
... nodules from pinpoint to child- head size . They exhibit some neoplastic qualities and contain normal structures in abnormal arrangements , which groups them between malformations and tumors . As hepatocellular adenomas consisting of ...
... nodules from pinpoint to child- head size . They exhibit some neoplastic qualities and contain normal structures in abnormal arrangements , which groups them between malformations and tumors . As hepatocellular adenomas consisting of ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall