The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 11
... observed fre- quently , but it is now realized that they represent artefacts brought about by anoxia or other alterations of the animals observed under such conditions . It is , therefore , now assumed that the bile canaliculi have a ...
... observed fre- quently , but it is now realized that they represent artefacts brought about by anoxia or other alterations of the animals observed under such conditions . It is , therefore , now assumed that the bile canaliculi have a ...
Page 57
... observed also as a result of a carcinoma of the pancreas head ( see page 148 ) and , to a lesser or negligible extent , of carcinoma of the body or tail . Pancreatic calculi , until recently considered comparatively rare but more ...
... observed also as a result of a carcinoma of the pancreas head ( see page 148 ) and , to a lesser or negligible extent , of carcinoma of the body or tail . Pancreatic calculi , until recently considered comparatively rare but more ...
Page 77
... observed , which indicates an inability of the liver to inactivate ( conjugate ) estrogens . In general , however , hepatic insufficiency in starvation is not severe enough to cause death by hepatocellular failure or hepatic coma . The ...
... observed , which indicates an inability of the liver to inactivate ( conjugate ) estrogens . In general , however , hepatic insufficiency in starvation is not severe enough to cause death by hepatocellular failure or hepatic coma . The ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall