The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 82
SECTION XVII - PLATE 20 COMPLETE OBSTRUCTION INCOMPLETE OBSTRUCTION . EXTRINSIC CANCER FIXING AND COMPRESSING DUCT INTRINSIC CANCER IMPACTED STONE WITH EDEMA STRICTURE BALL - VALVE STONE EXTRAHEPATIC BILIARY OBSTRUCTION 1 Mechanism ata ...
SECTION XVII - PLATE 20 COMPLETE OBSTRUCTION INCOMPLETE OBSTRUCTION . EXTRINSIC CANCER FIXING AND COMPRESSING DUCT INTRINSIC CANCER IMPACTED STONE WITH EDEMA STRICTURE BALL - VALVE STONE EXTRAHEPATIC BILIARY OBSTRUCTION 1 Mechanism ata ...
Page 83
BILE INFARCT The effects of biliary obstruction upon the liver itself , best observed in biopsy specimens , develop more rapidly in complete than in incomplete obstruction . The change first to appear is the accumulation of bile pigment ...
BILE INFARCT The effects of biliary obstruction upon the liver itself , best observed in biopsy specimens , develop more rapidly in complete than in incomplete obstruction . The change first to appear is the accumulation of bile pigment ...
Page 127
If the resulting obstruction remains complete for prolonged periods , the gallbladder gradually enlarges , its wall becoming thin and stretched . In earlier stages the lining epithelium secretes an increased amount of mucus .
If the resulting obstruction remains complete for prolonged periods , the gallbladder gradually enlarges , its wall becoming thin and stretched . In earlier stages the lining epithelium secretes an increased amount of mucus .
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall