The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Results 1-3 of 24
Page 47
From the quantity of bile pigment excreted, it has been calculated that under
normal conditions the rate of hemoglobin turnover amounts to 16 to 24 gm. per
day. It is thus evident that of the available pathways to break down hemoglobin
the ...
From the quantity of bile pigment excreted, it has been calculated that under
normal conditions the rate of hemoglobin turnover amounts to 16 to 24 gm. per
day. It is thus evident that of the available pathways to break down hemoglobin
the ...
Page 48
It is produced by a great variety of conditions, the common denominator of which
is a disturbance of the bile pigment formation or bile pigment flow. The degree of
pigmentation may vary from a scarcely visible to an intense discoloration, ...
It is produced by a great variety of conditions, the common denominator of which
is a disturbance of the bile pigment formation or bile pigment flow. The degree of
pigmentation may vary from a scarcely visible to an intense discoloration, ...
Page 159
... 136, 147 cystadenoma, 136, 145, 146 cysteine conjugation, 44, 64 cystic
disease, liver, 60 pancreas, see fibrocystic disease cystine deficiency, 77
cytochrome pigment, 88 cytologic smear examination, 53, 148 cytology, see cell
cytomegalic ...
... 136, 147 cystadenoma, 136, 145, 146 cysteine conjugation, 44, 64 cystic
disease, liver, 60 pancreas, see fibrocystic disease cystine deficiency, 77
cytochrome pigment, 88 cytologic smear examination, 53, 148 cytology, see cell
cytomegalic ...
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Contents
DISEASES OF THE LIVER continued | 3 |
PLATE PAGE NUMBER NUMBER 24 CarbohydratcLipid Disturbances | 86 |
Wilsons Disease | 87 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis alkaline phosphatase Amer anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholestasis cholesterol chronic cirrhosis clinical common bile duct connective tissue cystic duct cysts damage degeneration develop diagnosis dilated duodenal duodenum enlarged excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular histologic infection inferior intestinal intrahepatic islet jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobule lymph lymphatic metabolism metastases mucosa necrosis necrotic nodes normal onlv organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior primary hepatic protein rare regenerative nodules result septa serum sinusoids sometimes sphincter spleen splenic stage stones superior mesenteric surface surgical tion tumor urobilinogen usuallv usually vascular vessels viral hepatitis wall