The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 9
SECTION XV – PLATE 7 INTRAHEPATIC STRUCTURES Liver Cell Arrangement
Until about 10 years ago the standard ... models , such as the classical model of
Braus in which the presence of both plates and cords is demonstrated .
SECTION XV – PLATE 7 INTRAHEPATIC STRUCTURES Liver Cell Arrangement
Until about 10 years ago the standard ... models , such as the classical model of
Braus in which the presence of both plates and cords is demonstrated .
Page 10
It develops after birth , when the liver cell plates become convergent . ... alters the
positions of the liver cell plates which then appear almost to converge toward the
portal triad , producing the picIntrahepatic Biliary System ( Plate 9 , page 11 ) ...
It develops after birth , when the liver cell plates become convergent . ... alters the
positions of the liver cell plates which then appear almost to converge toward the
portal triad , producing the picIntrahepatic Biliary System ( Plate 9 , page 11 ) ...
Page 63
TWO - CELL - THICK PLATES REGENERATIVE NODULE CIBA FEATURES OF
HEPATIC REGENERATION AND ATROPHY MULTINUCLEAR GIANT CELLS
BILE DUCT PROLIFERATION . Cells of the liver plates constantly disappear and
...
TWO - CELL - THICK PLATES REGENERATIVE NODULE CIBA FEATURES OF
HEPATIC REGENERATION AND ATROPHY MULTINUCLEAR GIANT CELLS
BILE DUCT PROLIFERATION . Cells of the liver plates constantly disappear and
...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall