The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 10
The covering, however, is much thinner than that of the structures of the portal
triads, which explains the easy yielding of the hepatic vein system to
compression (see page 69). The ramifications of the hepatic vein and of the
portal vein cross ...
The covering, however, is much thinner than that of the structures of the portal
triads, which explains the easy yielding of the hepatic vein system to
compression (see page 69). The ramifications of the hepatic vein and of the
portal vein cross ...
Page 68
The septa, which have developed in different parts of the lobule, converge in
further stages of the process so as to create connections between portal triads
and central fields. These connections carry vessels which become anastomoses.
The septa, which have developed in different parts of the lobule, converge in
further stages of the process so as to create connections between portal triads
and central fields. These connections carry vessels which become anastomoses.
Page 92
If the microorganisms reach the sinusoids, they are either taken up and destroyed
by the Kupffer cells or they pass into the tissue spaces, whence they are drained
to the lymphatics in the portal triads to set up an inflammation within and ...
If the microorganisms reach the sinusoids, they are either taken up and destroyed
by the Kupffer cells or they pass into the tissue spaces, whence they are drained
to the lymphatics in the portal triads to set up an inflammation within and ...
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Contents
DISEASES OF THE LIVER continued | 3 |
PLATE PAGE NUMBER NUMBER 24 CarbohydratcLipid Disturbances | 86 |
Wilsons Disease | 87 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis alkaline phosphatase Amer anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholestasis cholesterol chronic cirrhosis clinical common bile duct connective tissue cystic duct cysts damage degeneration develop diagnosis dilated duodenal duodenum enlarged excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular histologic infection inferior intestinal intrahepatic islet jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobule lymph lymphatic metabolism metastases mucosa necrosis necrotic nodes normal onlv organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior primary hepatic protein rare regenerative nodules result septa serum sinusoids sometimes sphincter spleen splenic stage stones superior mesenteric surface surgical tion tumor urobilinogen usuallv usually vascular vessels viral hepatitis wall