The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 85
OSTEOMYELITIS PRIMARY AMYLOIDOSIS : AMYLOID DEPOSIT IN WALL OF
ARTERY - STAINED WITH CONGO RED BLOOD STREAM. ETIOLOGY :
CHRONIC SUPPURATION PULMONARY TUBERCULOSIS LUGOL ' S
SOLUTION ...
OSTEOMYELITIS PRIMARY AMYLOIDOSIS : AMYLOID DEPOSIT IN WALL OF
ARTERY - STAINED WITH CONGO RED BLOOD STREAM. ETIOLOGY :
CHRONIC SUPPURATION PULMONARY TUBERCULOSIS LUGOL ' S
SOLUTION ...
Page 97
It is this phase to which the name primary biliary cirrhosis ( see page 84 ) applies
and when cirrhotic transformation has just begun . In some patients with subacute
or chronic cholangiolitis ( primary biliary cirrhosis ) , the cholesterol rises to ...
It is this phase to which the name primary biliary cirrhosis ( see page 84 ) applies
and when cirrhotic transformation has just begun . In some patients with subacute
or chronic cholangiolitis ( primary biliary cirrhosis ) , the cholesterol rises to ...
Page 149
Because of the close contact with the duodenum and the lymphatic connection (
see page 30 ) , invasion of the duodenum with large tumors is so common that it
may be impossible to determine which was the primary site . Extension via the ...
Because of the close contact with the duodenum and the lymphatic connection (
see page 30 ) , invasion of the duodenum with large tumors is so common that it
may be impossible to determine which was the primary site . Extension via the ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
96 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall