The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 82
... produce jaundice , because the nonobstructed part of the liver takes over in a compensating fashion . The excretion of bile compo- nents other than the bile pigments , how- ever , may not be as readily compensated , and serum alkaline ...
... produce jaundice , because the nonobstructed part of the liver takes over in a compensating fashion . The excretion of bile compo- nents other than the bile pigments , how- ever , may not be as readily compensated , and serum alkaline ...
Page 92
... produce inflamma- tion in the tissue of the portal triads , espe- cially around the lymphatics . More often than by any other mode of transmission , bacteria reach the portal triads by way of the lymphatics and arterial blood , when the ...
... produce inflamma- tion in the tissue of the portal triads , espe- cially around the lymphatics . More often than by any other mode of transmission , bacteria reach the portal triads by way of the lymphatics and arterial blood , when the ...
Page 120
... produce this jaundice . Another theory takes into account that the newborn must dis- pose of the fetal hemoglobin F and that this process could be related to increased bilirubin production . But recent studies indicate that hemoglobin F ...
... produce this jaundice . Another theory takes into account that the newborn must dis- pose of the fetal hemoglobin F and that this process could be related to increased bilirubin production . But recent studies indicate that hemoglobin F ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall